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Jingdong book

常见病合理用药丛书：脂肪肝合理用药180问（第2版）

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Jingdong book

常见病合理用药丛书：脂肪肝合理用药180问（第2版）

Name: 常见病合理用药丛书：脂肪肝合理用药180问（第2版）
Brand: Jingdong book
SKU: 3117125071
Price: 24.16 USD
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什么叫脂肪肝？什么是脂肪性肝炎和肝纤维化？肝脂质沉积症与脂肪肝是否是一回事？什么是脂类和血脂？脂肪肝的发病状况如何？脂肪肝是如何形成的？脂肪肝的危害性如何？中医是如何看待脂肪肝的？为什么要重视脂肪肝的防治？哪些病因可以导致脂肪肝？诱发脂肪肝的危险因素有哪些？《常见病合理用药丛书：脂肪肝合理用药180问（第2版）》是“常见病合理用药丛书”之一，全书采用问答的形式，主要对脂肪肝的用药知识作了介绍。该书可供各大专院校作为教材使用，也可供从事相关工作的人员作为参考用书使用。

Content Description

《常见病合理用药丛书：脂肪肝合理用药180问（第2版）》以问答的形式，深入浅出地介绍了脂肪肝的相关知识，包括脂肪肝的概念、病因、病理、实验室和影像学检查、诊断及治疗，重点介绍了防治脂肪肝药物的合理应用。全书内容丰富，通俗易懂，适合于脂肪肝患者及其家属阅读参考。

Catalogue

基本知识
1．什么叫脂肪肝？
2．什么是脂肪性肝炎和肝纤维化？
3．肝脂质沉积症与脂肪肝是否是一回事？
4．什么是脂类和血脂？
5．脂肪肝的发病状况如何？
6．脂肪肝是如何形成的？
7．脂肪肝的危害性如何？
8．中医是如何看待脂肪肝的？
9．为什么要重视脂肪肝的防治？
10．哪些病因可以导致脂肪肝？
11．诱发脂肪肝的危险因素有哪些？
12．为什么说肥胖是脂肪肝的主要诱因之一？
13．营养不良是否会诱发脂肪肝？
14．常见的导致脂肪肝的化学因素有哪些？
15．妊娠脂肪肝是怎么回事？
16．哪些药物可以引起脂肪肝？
17．为什么糖尿病患者容易出现脂肪肝？
18．高脂血症与脂肪肝常常同时存在吗？
19．脂肪肝与代谢综合征关系如何？
20．肝炎患者为何谨防脂肪肝？
21．病毒性肝炎与脂肪肝有关吗？
22．脂肪肝会遗传吗？
23．儿童会不会得脂肪肝？
24．饮酒与脂肪肝的关系如何？
25．饮酒的量和方式与脂肪肝的关系如何？
26．饮酒对人体还有哪些危害？
27．脂肪肝与肝硬化及肝癌有何关系？
28．哪些人更容易患脂肪肝？
29．患了脂肪肝的人会有哪些临床表现？
30．脂肪肝常与哪些疾病合并存在？
31．根据脂肪肝的轻重可以分几种类型？
32．脂肪肝患者应该重点了解哪些信息？
33．诊断脂肪肝的主要临床依据是什么？
34．脂肪肝的实验室检查主要有哪些内容？
35．如何早期发现脂肪肝？
36．如何判断酒精中毒？
37．酒精性肝病常见的生化指标异常有哪些？
38．非酒精性脂肪性肝病有哪些检查指标？
39．反映肝脏炎症损伤的指标主要是哪些？
40．肝脏合成功能受损主要表现在哪几个指标异常？
41．脂肪肝还可以出现哪些指标异常？
42．如何判断肝脏纤维化或者肝硬化？
43．发生严重肝损伤如何判断病情轻重？
44．为什么说超声诊断是脂肪肝的首选诊断方法7
45．脂肪肝的声像特点如何？
46．何为脂肪肝的超声定性与定量诊断？
47．肝脏CT检查对脂肪肝的诊断价值如何？
48．脂肪肝的CT影像特点如何？
49．何为脂肪肝的CT定性与定量诊断？
50．酒精性脂肪肝的CT扫描有何特点？
51．肝硬化的CT影像学特点如何？
52．磁共振（MRI）如何诊断脂肪肝？
53．局灶脂肪肝与肝癌和转移瘤如何鉴别？
54．放射性核素扫描可以诊断脂肪肝吗？
55．哪些脂肪肝患者需要做肝活检？有何意义？
56．肝活检的禁忌证有哪些？
57．脂肪肝的病理学特点和分类如何？
58．酒精性脂肪肝有哪些病理学特点？
59．脂肪性肝纤维化和肝硬化的组织学诊断依据是什么？
60．病理学诊断为什么也有不足之处？
61．酒精性脂肪性肝病与非酒精性脂肪性肝病的区别是什么？
62．如何鉴别单纯性脂肪肝与其他原因的脂肪肝？
63．脂肪肝，特别是没有症状的脂肪肝是否需要治疗？
64．脑病脂肪肝综合征以及肝豆状核变性脂肪肝的防治原则及目的是什么？
65．脑病脂肪肝综合征以及肝豆状核变性脂肪肝的治疗手段有哪些？
66．为什么说调整饮食热卡对于脂肪肝的治疗十分关键？
67．脂肪肝患者如何正确进食？
68．快速减轻体重是否对脂肪肝患者有益？
69．如何根据体重变化调整进食量？
70．医务人员对脂肪肝患者的作用如何？在脂肪肝治疗开展之前，医务人员应做哪些工作？

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Book Abstract

（1）食物中脂肪供应过多。长期和大量进食高脂肪、高胆固醇饮食导致外源性脂肪大量增多，结果从小肠吸收后进入血液中的乳糜微粒增加，肝脏从血液中摄取或合成甘油三酯相继增多，尽管肝细胞极低密度脂蛋白合成亦相应增多，但仍不足以将过多的甘油三酯转运人血，导致甘油三酯在肝细胞内异常沉积，形成脂肪肝。
（2）血浆游离脂肪酸过多。生长激素、糖皮质激素等可以激活外周脂肪组织的脂肪酶，导致机体贮存的脂肪水解为甘油和游离脂肪酸，大量游离脂肪酸进入血液。与体重正常者相比，肥胖和2型糖尿病患者体内脂肪酸释放入血明显增加，血浆游离脂肪酸显著升高，肝细胞从血液摄取游离脂肪酸增加，用于合成甘油三酯的脂肪酸相继增多。
（3）肝细胞脂肪酸氧化利用减少。脂肪酸在肝细胞内如能得到充分的氧化，转变成能量，肝细胞合成甘油三酯的量就会减少。当肝细胞对脂肪酸氧化障碍时，肝细胞必将通过加速合成甘油三酯以代谢脂肪酸。大量饮酒以及服用某些药物（如胺碘酮）可抑制肝细胞线粒体脂肪酸的氧化能力，使得甘油三酯合成增加，从而易于形成脂肪肝。
（4）肝内极低密度脂蛋白合成及排泄障碍。肝细胞合成的甘油三酯主要是与载脂蛋白结合成极低密度脂蛋白的形式转运人血。长期饥饿、消化吸收不良、低蛋白饮食等原因使蛋白质摄人不足、氨基酸供应减少或必需脂肪酸缺乏，导致合成磷脂和脂蛋白的原料不足，进而影响脂蛋白的合成；肝细胞炎症、坏死可使甘油三酯与脂蛋白结合障碍；四环素以及某些抗肿瘤药物可使载脂蛋白合成减少；先天性载脂蛋白合成障碍以及长期服用秋水仙碱，可引起肝细胞微管结构及功能受损。以上原因均可导致肝细胞内合成的甘油三酯不能顺利转运入血，尽管甘油三酯合成并未增加，但肝细胞内仍会出现脂肪蓄积。
总之，任何原因引起肝细胞内甘油三酯合成和极低密度脂蛋白分泌之间失去平衡，均可导致脂肪在肝细胞内异常沉积，进而形成脂肪肝。
……

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